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Smoking My research on cigarettes began in 1998. I called Brown and Williamson and personally requested a list of ingredients. You know, it behooves me that people (smokers) could smoke a product that does not even list the ingredients on the package, but when you understand how many ingredients go into cigarettes, then it makes sense and becomes understandable why the ingredients are not on the box. There are over 700 ingredients in one single cigarette, including a new strain of genetically engineered tobacco with a nicotine level ten times higher in potency than normal nicotine; caffeine, sugar, salt, rum, alcohol, codeine, menthanol (rocket fuel), and uric acid (yes, they even use piss or pee). Most smokers don’t know it, but the cigarette papers are dipped in opium (the true addictive agent of cigarettes). However, one ingredient among the other 700+ really stands out. This ingredient is “Civet Cat Absolute.” Now what in the hell is civet cat absolute? Well, as a researcher, I did the research and was shocked and amazed to find out that civet cat absolute is an umbrella term for a liquid that is obtained from the rectum of the civet cat. What is this ingredient used for? One reason is that it is a pheromone. Pheromone affects behavior in certain mammalian species. Why does pheromone have to be in cigarettes? Your guess is as good as mine. Every time you put a cigarette into your mouth, symbolically you are placing a white male phallic symbol into your mouth. It’s a power thing. Yes, cigarettes represent the white male phallic (cigars represent the black man’s penis). Cigarettes were created in part by Sigmund Freud’s nephew in a campaign to make women feel liberated and powerful. Power is a male attribute. In addition to the power thing, cigarettes also help women to lose weight by seriously impairing appetite. Why is Virginia Slims marketed only to women under the slogan, “It’s a woman thing”? Because subliminally, the initial message is: Virginia (a female name) is slim (skinny). The ultimate subliminal message (to women) is: Virginia Slims can help you stay slim by smoking this particular brand. You see, obesity, weight loss, being thin or slim is a female preoccupation in this country, hence the slogan, “It’s a woman’s thing.” “He who smokes is a walking billboard advertising himself as fool to the world!” For the sake of our environment and human health, especially our youth – stop being a weakling and an idiot who smokes. The law of reciprocity is nowhere to be found in smoking cigarettes or anything else. product(ciga-quit): http://www.dherbs.com/catalog/product_info.php?cPath=43&products_id=47 pro solution pills side effects surgical pennis enlargement pennis enlargement pump pennis enlargement pump do penis elargement pills really work penis elargement doctor free penis elargement technique pennis enlargement pills

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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. 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THE BICYCLE OF LEONARDO DA VINCI: - “… a man who was at once an artist, an inventor, a scientist - and who saw no contradiction between these diverse realms. In his unceasing quest for truth, Leonardo explored every branch of the sciences known to his age and proved to be far ahead in many respects in his precise observations, his striving for sound methodology and measurement, and the value he placed on empirical proof. 'No human investigation', he wrote, 'can be called true science without going through the mathematical tests… the sciences which begin and end in the mind cannot be considered to contain truth, because such discourses lack experience, without which nothing reveals itself with certainty." (57) This quote is from a semi-whitewash of a life full of conflict, especially with the church. They should emphasize the oppressive control over thought and creativity the church of Leonardo's day had over all sciences. In fact there was only one science - philosophy which in actual fact was just theology. So Leonardo's words take on a different meaning than they suggest. It is even more important than you might imagine because Leonardo was the head or ‘Nautonnier’ of the Priory of Sion. The authors are from Catholic Universities mostly, so we must forgive them when they claim to have fostered Leonardo's genius when in fact they did the opposite and it isn't till near the end of the book they note he was charged with being homosexual by these Inquisitorial suppressors of soul and thought. There is a whole specialty of study devoted to Da Vinci's work but little truth and a lot of propaganda. Few are they who admit both he and his mentor were alchemists and the horrors of a life hidden behind lies and half-truths that resulted from the fear of being found out. It is a theme of many of the great scientists and their lives, and little credit is given to the courageous men and women who toiled to understand and the truth they shared is available. Why? Do we still fear others might do what they did or think? Think for themselves and question the authority which seeks to credit their approach to knowing. The Scientific Method of observation and conclusion is said to have been discovered by Bacon. There is some truth to that, but which Bacon? Some say Francis and there is no truth in that even though he pretended to be an alchemist in his Rosicrucian or Masonic circles including John Dee and Ashmolean types. The better scholars attribute it to Roger Bacon who actually was an alchemist and spent a great deal of his life in dungeons after having ferreted himself away in the church as a monk for a long time. Metaphysics and independent thought can be a dangerous business and there are many current authors whose work is relegated to obscurity unless they learn how to appease the 'normative' or oppressive mindset. The sad part is that there is no benefit to anyone by such oppression. Not only is there abundance and creativity but those who know the soul will not NEED to clothe it in grandiose raiment. They will gladly work for the joy of utilizing their potential for the benefit of mankind rather than seeking to destroy others and life in general. It is interesting that Napoleon (a Merovingian/Mason) made sure to get all of Da Vinci's work that was around when he conquered Milan, but I there is some of it that wasn't discovered until after that and it is most interesting. Da Vinci was a Johannite like Newton (another alchemist, whose Principiae Mathematica had a brief comment at its' start - 'This is much more than I should say; and much less than there is!'). Johannites believe the true prophet in Biblical times was John the Baptist and we are continuing to seek other Benjaminite and Masonic connections with him despite a sense that both Jesus and John the Baptist were operating from the same source. Would Napoleon have destroyed any Johannite writings if the Merovingian House of David and Judah or family of Jesus were concerned about Da Vinci's fame and credibility or writings? We cannot say such a thing and we aren't sure John wasn't a favourite of theirs as well. The troubling thing about this book is the presentation of Da Vinci as a semi-competent who needed a Franciscan monk’s help. We like Francis of Assisi and don't doubt the church was watching over Leonardo just as they did all artists who were producing God's work. Yes, the church claimed all creative work was their property! They also controlled education just as they have in many places until this very day. Most likely Luca Pacioli (Franciscan) was under directions to keep Da Vinci in line and to make such things as we will see, remain hidden from the public. Da Vinci was before Galileo and you can be sure he would have met the usual gruesome heretical stake or fire after a few parts were titillated or cut along the way. These are the words of the Catholic University academic which give a little insight to the conflict Da Vinci faced during these oppressive times when thought was even less well managed than today. "Leonardo and Luca Pacioli worked together for many years, and as we have seen, the intervention of Master Luca was decisive. Nevertheless there existed between the two categories a social and hierarchical conflict, even if no one had ever placed in doubt the supremacy of the liberal arts as the only depositories of true science. And the one who rose up resolutely against the exclusion of the mechanical arts from the sphere of science {Such deceit to call what the church allowed people to think of, as science.}, or as it was then known, 'philosophy' was Leonardo." (58) The best part of the whole book for me is the bicycle. It has a chain drive mechanism that looks like a 19th century model of something he would have seen in the future. His representation is not engineered so perfectly that he would have been able to inspect it however. It had no steering mechanism, for example. On top of the original drawing that was discovered long after the death of all parties is a childish drawing of a penis to look like a cat with legs. Still they don't actually use words to say he was 'gay' and we know he would say something about this if he was alive today. It is a certain thing about propaganda - great people become more manageable after they die - you can 'spin' their image to your hearts desire. In the case of Jesus they make him over every century! I guess all the priests abusing children across North America and the court awards have made them a little sensitive - can you say risk management? Can you say hypocrites? They still exclude 'gays' from all sorts of things and marriage is just one of them. Maybe this is the area 'master' Luca was most instructive to Da Vinci?! It is an area of their expertise but what Da Vinci was able to produce, is not. "While they recognize the unmistakable nature of this machine, the few scholars who have examined the drawing are decidedly reluctant to admit its antiquity. Since the application of the chain-drive goes back only to the end of the 19th century, they propose a dating of the drawing within the early years of the present century. Such a hypothesis, however, collides with insurmountable difficulties: (1) The page in question remained hidden for almost four centuries, and it is unimaginable that 70 or 80 years ago a boy would have obtained from the directorship of the Biblioteca Ambrosiana the permission to view the codex, detach one or two of the pages, and then draw upon them and glue them back again. (2) Even in that case, he would have drawn a bicycle of a type then existent, not one of wood with wheelbarrow wheels, {And this imaginary child the academics try to say drew the cat/penis, would have been a pretty smart kid to know the name of Leonardo's pupil/model/lover. There is a small matter of his signature.} no means of steering, and the teeth of the central gear so squared off that they could not be fitted to the chain. (3) The odd toothed wheels and the chain coincide exactly with those drawn by Leonardo in Codex Madrid 1, folio 10 recto. (4) We cannot separate the bicycle from the other drawings visible in folios 132 verso and 133 verso of the Codex Atlanticus. Actually they were drawn when the pages were united at the two halves of one page. Reuniting them, we see that another hand has drawn, also in pencil and from left to right, two pornographic drawings obviously meant as a joke, over which, on the right side, is clearly written 'salaj', that is, Salai, the name of Leonardo's pupil, model and servant...To become a reality, the idea required the final solution of some challenging problems such as the matter of steering and the adaptation of the large squared off teeth with their jutting comers to the links of the chain… Salai entered Leonardo's house in 1490, aged 10.” (59) How did Leonardo draw about half of the design of such as bike? The best answer is that he looked into the 'Philosopher’s Stone' of his patron/alchemist or he developed one of his own like Nostradamus. It is not an easy thing to do, but I think he could have done it. If he actually traveled through time he would have been able to draw a bike and many other things like his famous helicopter. When looking into a 'Stone' there are many confusing images of possible future and jumbled realities. We know that many within the Catholic Church have studied these things (e.g. Aquinas) and that they know about the 'Stone'. In recent times a Catholic cleric developed a Chronovisor in conjunction with top physicists including Enrico Fermi, according to one report I have seen. We know they would have been very concerned about Leonardo telling the world about many things he knew. The Gnostics were heretics because they said things like: 'The Original Sin that separates us from God is Ignorance!' Who is passing on whether or not you are a heretic because you believe in the possibility of a 'Stone' today? Yes, most of society! Few have any idea of what an alchemist is or what contributions they've made to humanity, the quantum physicists were called 'atom-mysticists' when they spoke of the things chaos science and alchemy has studied for over 13,000 years. This is why we don't need an Inquisition. Because the media and education system is more effective! The titles and grandiloquent appointments of the egotistical chest-beating ecclesiasts who follow in the footsteps of the money- changers whose tables Jesus over-turned in the Roman-backed Temples are so fantastic that any impressionable person can easily think THEY must know! This is the technique of 'The BIG lie!' If you make an ass of your true self (the soul) by saying you are 'Holy' and an 'Emperor', it is no matter. Just continue to add all the nations and states you have under your control. Say you are the sole representative of God on earth, and that the universe revolves around you. It might work as well as saying disease comes from 'sins and demons' and 'women are the harbingers of THE Original Sin'. Actually any human is able to know what Jesus called 'the living father within'. Are we really out of the influence of the 'Dark Ages'? I'd like to debate that with the Pope or some other proselyte. plastic surgery penis elargement penis enlargment product top rated penis enhancement pills online vig rx penile enlargment before and after photo compare penile enlargment pills permanent penis enlargment penis elargement testimonials free exercise tip for penis enlarement

The prostate is a gland in the male reproductive system. It is located just below the bladder and wraps around the urethra. The prostate gland measures 3-4 cm long and 3-5 cm in width. On average, the prostate gland weighs 20 grams. The prostate consists of approximately 30% muscular tissue while the remaining is glandular tissue. Seminal vesicles are attached to the prostate and produce material that mixes with prostatic fluid to form semen. The tubes from the testicles carry sperm to the prostate, which mixes with the seminal fluid and is ejaculated during orgasm. The prostate may increase in size as age progresses. This condition is called benign prostatic hyperplasia (BPH). More than 40% of men have an enlarged prostate by the age of 70. Enlargement of the prostate causes it to press against the urethra and weaken the flow of urine. An increase in size may indicate the condition of benign prostatic hyperplasia or a urologic condition. It need not necessarily indicate the growth of cancer cells. Benign prostatic hyperplasia does not increase the risk of prostate cancer but indicates the possibility of occurrence. Three common diseases of the prostate are benign prostatic hyperplasia (BPH), prostates and prostate cancer. Each condition affects the prostate differently. The male sex hormone, testosterone, plays an important part in the normal growth and function of the prostate gland. The testicles produce testosterone and is a concern for those diagnosed with hormone-dependent prostate cancer. As long as testosterone is produced, prostate cancer is very likely to grow and spread throughout the body. The prostate has various concentric zones, known as the anterior fibromuscular stroma, peripheral zone, central zone and transition zone. A doctor is able to examine the peripheral zone by inserting a finger in the rectum. Benign prostatic hyperplasia develops in the transition zone and grows in size. The anterior fibromuscular stroma is the anchoring point of the urethra sphincter. It does not contain any glands and hence cancer or enlargement does not usually develop there. It is essential to contact a urologist in case one experiences the symptoms to ensure early preventive care. surgical penis enlargment penis enlagement patch vig rx store enlargement penis pills vimax vimax forum natural penis enargement do penile enlargement pills work truth about penis enlarement free exercise tip for penis enlarement